Articles For Doctors
Eyelights & The Brain

When light enters the eye information travels from the retina to the lateral geniculate nucleus (LGN) located in the thalamus of the brain. There are two geniculate nuclei, each comprised of 6 layers: two magnocellular layers and 4 parvocellular layers. Magno pathways have large cells that carry out fast processes for perceiving position, motion, shape, and low contrast. The magno cells form the major input to the dorsal stream (parietal lobes) or "where or how" pathway. Parvo pathways have smaller cells that carry out slower processes for perceiving still images, color, detail, and high contrasts. The parvo cells form the major input to the ventral stream (temporal lobes) or "what" pathway.

Because eye dominance sets in and information becomes suppressed, only 6 of the 12 layers are being excited. Therefore, only 50% of the thalamus is being stimulated. When Eyelights are applied to the non-dominant eye in a monocular fashion excitation occurs within the 6 lesser functioning layers, resulting in stimulation of the entire thalamus.





An excitatory barrage then travels to the mesencephalon, the most metabolic area of the brain, causing an increase in cellular activity. When these cells become excited they metabolize glucose for energy and produce new proteins in order to stay healthy, creating a cell with more stamina and endurance. The excitatory barrage travels also to the parietal, temporal, and occipital lobes of the brain, while collateral fibers lead to the pineal gland, pituitary gland, and hypothalamus.

Neurotransmitters are naturally occurring chemicals responsible for communication of information between neurons in the brain and between the brain and organs of the body. A chemical imbalance occurs when an irregularity exists in the production, absorption, or re-absorption (reuptake) of neurotransmitters like serotonin and dopamine. It is thought that a continuous state of stress or agitation experienced over extended periods of time can force the brain to cycle through important neurotransmitters too quickly, creating an imbalance.

Research has shown that light projections into the eye can have a profound effect on the hormonal system, emotions, stress levels, sleep, brain function, and many other aspects of a patient's biochemistry. One study showed remarkable changes in the concentration of neurotransmitters in the cerebro-spinal fluid. [1]

Serotonin is a major neurotransmitter found in cardiovascular tissue, the peripheral nervous system, blood cells, and the central nervous system. 90% of serotonin is in the intestine and the rest in blood platelets and the brain. Although the CNS contains less than 2% of the total serotonin in the body, serotonin plays a very important role in a range of brain functions. Within the brain, serotonin is localized mainly in nerve pathways emerging from the raphe nuclei in the pons, where A7 and A8 cells produce serotonin. These serotonergic pathways spread extensively throughout the brainstem, cerebral cortex, and the spinal cord.

In addition to mood control, serotonin has been linked with a wide variety of functions, including the regulation of sleep, pain perception, body temperature, blood pressure, and hormonal activity. Outside the brain, serotonin is particularly involved with the gastrointestinal and cardiovascular systems.

In order to stimulate serotonin production, Eyelights will be worn with the top row of lights blinking brighter.

Dopamine is critical to the way the brain controls our movements and is concentrated in groups of neurons called the basal ganglia. The basal ganglia are a collection of neurons deep to the white matter of the cerebral cortex. Dopaminergic neurons are widely distributed throughout the brain via three pathways. Movement control also involves the interaction of many other brain regions, including the motor cortex, the thalamus, the cerebellum, and a large number of neuron groups located within the mesencephalon and brain stem. Within the mesencephalon lie the substantia nigra and the A9 and A10 dopamine cell groups. Loss of dopamine neurons in the substantia nigra, which connects with the basal ganglia, is a major factor in Parkinson's disease, where a person loses the ability to execute smooth, controlled movements.

Disruption of the dopamine system has also been linked to psychosis, schizophrenia, and addiction. Drugs used to enhance the action of dopamine have been known to cause sleep attacks, hallucinations, dizziness, headaches, and fatigue. Anti-psychotic drugs, which block dopamine receptors, can even cause symptoms normally associated with Parkinson's such as tremors, dystonia, and the slowing down of facial expression and body movements.

In order to stimulate dopamine production, Eyelights will be worn with the bottom row of lights blinking brighter.

[1] Effects of the Lumatron Upon Neurochemicals: Dr. Norman Shealy , M.D., Ph.D





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